Preliminary experiments indicate conolidine could inhibit certain ion channels, decreasing neuronal excitability and restricting ache signals. This system is particularly applicable in neuropathic discomfort, where by abnormal signaling brings about persistent discomfort. Additionally, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to discomfort pe... https://jamesw950qjy6.onzeblog.com/profile